An ambitious study of people with LongCovid, the mysterious, disabling symptoms that can trail a SARSCoV2 infection, has turned up a host of abnormalities in their blood.
An ambitious study of people with Long Covid, the mysterious, disabling symptoms that can trail a SARS-CoV-2 infection, has turned up a host of abnormalities in their blood. The clues add to a body of evidence hinting at drivers of the condition and potential treatments worth testing. They also suggest that, as many scientists and patients have suspected, Long Covid shares certain features with myalgic encephalomyelitis/chronic fatigue syndrome , another condition thought to follow an infection.
Other groups studying Long Covid patients have reported similar results this year, including in a January, and reactivation of viruses in patients with neurological issues. Collectively, these data “make me think about what other drugs we can test,” such as virus-directed antibodies or targeted anti-inflammatories to tame the immune system, says Emma Wall at University College London and the Francis Crick Institute, who co-leads a large trial of potential Long Covid therapies.
The low cortisol levels in the Long Covid patients, about half of normal levels, aren’t a total surprise: Symptoms such as fatigue and muscle weakness are associated with less of the hormone. The cause remains a mystery. ACTH, a hormone made by the pituitary gland that controls cortisol production, was at normal levels in the Long Covid group.
This battle would produce chronic inflammation, which matches many Long Covid symptoms. By measuring levels of antibodies against viral proteins released in the blood, the study also noted reactivation of Epstein-Barr virus and other herpesviruses whose genes can sit dormant inside infected cells for extended periods.
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