SARS-CoV-2 lineage EG.5.1 has an advantage at evading neutralizing antibodies. When vaccinated or infected, our immune system creates antibodies that target the spike protein of SARS-CoV-2, blocking the virus from entering and multiplying inside cells. In turn, the virus evolves mutations that redu
Research indicates that the newly emerged Eris lineage of SARS-CoV-2, characterized by moderate antibody escape capabilities, is responsible for its increasing spread; however, the forthcoming vaccines based on the XBB.1.5 lineage are anticipated to be effective against it. The severity of illnesses caused by these variants remains unchanged.When vaccinated or infected, our immune system creates antibodies that target the spike protein offrom entering and multiplying inside cells.
After exhausting years of the pandemic with multiple waves of infections caused by ever-changing virus variants and corresponding hospitalization rates, the situation has significantly improved by now. Large waves of infections outside the cold and wet seasons are not occurring. This success is largely attributed to the rapid development of vaccines.
A part of our immune protection relies on neutralizing antibodies that are produced by the cells of our immune system after vaccination or infection. Neutralizing antibodies attach to the spike protein of SARS-CoV-2, preventing the virus from entering into our cells. This mechanism is also referred to as neutralization.
“Furthermore, mutations can enhance the transmissibility of SARS-CoV-2 variants by, for example, improving the binding of the spike protein to the cellular receptor ACE2,” says Markus Hoffmann, the leading scientist behind the study. “We tested how effectively the Eris sublineage EG.5.1 can enter host cells and how efficiently it is neutralized by antibodies in the blood of vaccinated individuals without a SARS-CoV-2 infection and those with a SARS-CoV-2 infection. During this process, we found that, in comparison to other currently circulating SARS-CoV-2 lineages, EG.5.1 does not possess an advantage in infecting host cells.
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